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In human studies the correlation of resistin and nutritional status is less clear, but increased resistin expression has been correlated with inflammatory markers, coronary artery disease, cardiovascular disease, asthma, and rheumatoid arthritis [126, 127]

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For example, when cultured in selenium-deficient conditions, Jurkat cells (a human T cell model) exhibit increased spontaneous cell death [18], due to decreased expression of selenium-dependent antioxidant enzymes (i.e., selenoproteins) such as glutathione peroxidases (GPx) and thioredoxin reductases leading to excessive oxidative stress and eventually cell death

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