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doi: 10.1038/S41574-019-0280-9 47 HompeschMJones-LeoneACarrMCMatthewsJZhiHYoungMet al

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The central role of mitochondrial dysfunction and nuclear DNA fragmentation in APAP-induced liver injury in the mouse model is also replicated in humans as illustrated by the detection of a mitochondrial matrix enzyme, glutamate dehydrogenase, mitochondrial DNA and nuclear DNA fragments in patients with APAP overdose [155]