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Cell Reports Abstract FTY720 requires vitamin B12-TCN2-CD320 signaling in astrocytes to reduce disease in an animal model of multiple sclerosis Highlights CD320 expression is suppressed in MS and EAE astrocytes S1P 1 inhibition upregulates CD320 expression in astrocytes Fingolimod and sphingosine directly bind to TCN2 Fingolimod-bound TCN2 promotes CD320 internalization in astrocytes Summary Vitamin B 12 (B 12 ) deficiency causes neurological manifestations resembling multiple sclerosis (MS)

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Having a high MCV may indicate that someone is consuming high levels of alcohol or has alcohol use disorder (AUD)