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(2018), both the initial TL and the life-course rate of telomere shortening are highly plastic and sensitive to early-life conditions

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Hence, GSH depletion is not a relevant signaling mechanism after exposure to low, subtoxic ITC doses while treatment with cytotoxic ITC concentrations potentially triggers low GSH responsive genes ( Application of 14 C-labeled SF to animal cell lines revealed that extracellular SF was rapidly depleted in the medium and that intracellular enrichment of SF was driven by conjugation of SF to GSH