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Conversely, the ability of A2A receptor blockade to enhance effector immunity, as utilized in cancer immunotherapy, underscores the context-dependent nature of this pathway and emphasizes the necessity for thorough safety evaluations when considering A2A receptor agonists for the treatment of inflammatory eye diseases ( In summary, evidence from uveitis and EAU indicates that impaired A2A receptor function may hinder TIGIT+ regulatory T cells, affecting immune regulation in the eye

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