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In scientific literature, GHK-Cu is studied as a signaling-associated peptide complex and is frequently investigated in cell-culture and preclinical models examining gene-expression regulation, extracellular matrix biology, cellular communication pathways, and copper-dependent regulatory processes

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Multiple stress responses, including autophagy, cell-cycle arrest, senescence, apoptosis, programmed necrosis and inflammation, have important roles in the pathogenesis of cisplatin nephrotoxicity