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highlighted that KRAS mutation increased IGF1R expression at both the mRNA and protein levels through Y-box-binding protein-1 (YB-1) upregulation which was then validated as a miR-137 target gene

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By contrast, in the experiments in which the course of ischemia was not reversed, decreased NO levels in the colon tissue appeared as an immediate heavy loss of endothelial cells from the vascular wall, which resulted in a lower eNOS production ability[42]