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Innate immunity in diabetic kidney disease | Nature Reviews Nephrology

Furthermore, in a study using autologous recipients, one could not determine whether protective compensatory mechanisms in the plasma of G6PD-deficient subjects could compensate for their poor redox status, as previously reported (55), or whether the difference in PTR is due to an intrinsic defect of G6PD-deficient RBCs as opposed to extrinsic effects of G6PD-deficiency on RBC clearance

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Reads were trimmed using Trimmomatic (version 0.39) with the following parameters: LEADING:20, TRAILING:20, SLIDINGWINDOW:4:15, and MINLEN:20, based on Illumina adapter sequences (CTGTCTCTTATACACATCT, AGATCGGAAGAGCACACGTCTGAACTCCAGTCA, AGATCGGAAGAGCGTCGTGTAGGGAAAGAGTGT)